I do strength training 3 times a week with some GPP/cardio and my resting heart rate is usually above 80 bpm. However, I do not experience any symptoms. So my question is, is it generally true (or is it a myth) that higher-than-average resting heart rates increase chances of heart disease, even for those who are physically active? Please note I am not worrisome and freaking out, but simply curious for your expert opinion.
I met all the conditions and tested with results of 83 bpm (using Android ECG feature) and ~80 bpm manually. I am completely relaxed. Also, I am male, 27 years old. Without presence of any symptoms, I am not sure whether or not I should consider this an issue I should remedy, or if there is any connection with my training.
I probably wouldn’t trust a smart phone app over an actual ECG (3 lead or better) for something like this and I it’s difficult to palpate one’s one resting HR as well. Still, I would agree > 80 bpm would probably cause me to address it with conditioning.
I’m not sure what you’re asking with “How do 60-80bpm resting HR individuals die?” They die from all sorts of things- cancer, infections, MVA’s, etc.
That said, the data on resting heart rate does not reliably predict death from CVD.
Weird, but data nonetheless.
How does someone lower his RHR, should I ask my doctor for a Ăź blocker?
More conditioning, weight loss if needed, etc.
Turns out, many people without a sufficient aerobic base will not develop said aerobic base by merely squatting.
I don’t think I understand, sorry for being dense, if I am gonna die from pancreatic cancer and I have higher than normal (what is normal? last time I checked everything under 100 was fine) RHR. What will using all means to lower my RHR gonna do for me? Will it prevent me from getting cancer?
howdy, I have asthma, I dont know if correlates but my rest rate generally is 55-60 BPM, its normal have a litlle bit more heart rate on people with asthma? I use LABA twice a day.
I’ll consider that, as long as there’s correlational data that chilling out reduces all-cause mortality and that the mechanism is indipendent from the reduction in RHR.